World Journal of Nephrology and Urology, ISSN 1927-1239 print, 1927-1247 online, Open Access
Article copyright, the authors; Journal compilation copyright, World J Nephrol Urol and Elmer Press Inc
Journal website http://www.wjnu.org

Case Report

Volume 5, Number 1, March 2016, pages 11-15

Acute Renal Infarction Pathogenesis and Atrial Fibrillation: Case Report and Literature Review

Figures

Figure 1.
Figure 1. CT scan of the abdomen showing bilateral kidney infarctions.
Figure 2.
Figure 2. A diagram of the most common pattern of arterial supply to the kidneys demonstrating the main renal artery, anterior and posterior branches, and five segmental arteries. MRA: main renal artery; PD: posterior division; AD: anterior division. Segmental arteries are indicated by A (apical), U (upper), M (middle), L (lower), and P (posterior). Modified from Graves FT. The anatomy of the intrarenal arteries and its application to segmental resection of the kidney. Br J Surg 1954;42:132.
Figure 3.
Figure 3. An allograft kidney from a 58-year-old obese man with a past medical history of hypertension and diabetes mellitus, who expired due to acute myocardial infarction and was found to have ARI on autopsy.
Figure 4.
Figure 4. Gross image of the bivalved kidney with cortical infarcts secondary to arterial thrombus, largest infarct (black arrow) measuring 5.0 cm.
Figure 5.
Figure 5. Microscopic image of an arterial thrombus showing a vessel completely occluded by the thrombus (black arrow) and the resulting renal infarct (in and around circle) (from Fig. 4) (H&E, × 2.5).